Somatic mutations in chromatin remodeler genes could modify the epigenomic landscape of CLL, but they are uncommon Within this malignancy when compared with other lymphoid neoplasms. CHD2
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Environmental or self-antigens and homotypic interactions induce BCR and Toll-like receptor (TLR) signaling, amplifying the response of CLL cells to other signals through the microenvironment and increasing the activation of anti-apoptotic and proliferation pathways.
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